MALADIE DE CHURG ET STRAUSS PDF

Alternative treatments include plasma exchange, 3 interferon alfa, 4 and intravenous immunoglobulin. A 37 year old woman case 1 presented with an 8 month history of nasal congestion, hearing loss, lymphadenopathy, rash, breast inflammation, peripheral neuropathy, abdominal pain, malaise, and weight loss. Tachydysrhythmias with poor left ventricular function on echocardiogram suggested cardiac vasculitis. Bone marrow nasal and skin biopsies demonstrated prominent eosinophil infiltration, and a chest computed tomography scan showed pulmonary infiltrates.

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Alternative treatments include plasma exchange, 3 interferon alfa, 4 and intravenous immunoglobulin. A 37 year old woman case 1 presented with an 8 month history of nasal congestion, hearing loss, lymphadenopathy, rash, breast inflammation, peripheral neuropathy, abdominal pain, malaise, and weight loss.

Tachydysrhythmias with poor left ventricular function on echocardiogram suggested cardiac vasculitis. Bone marrow nasal and skin biopsies demonstrated prominent eosinophil infiltration, and a chest computed tomography scan showed pulmonary infiltrates. There was a peripheral eosinophilia 7. CSS was diagnosed. Five months after a third course of alemtuzumab her disease relapsed, presenting with malaise, nasal obstruction, asthma, and peripheral neuropathy.

A 35 year old woman case 2 with known CSS presented in January with relapsing disease reflected by malaise, fatigue, asthma, peripheral neuropathy, night sweats, polyarthritis, multiple subcutaneous nodules, and an erythematous rash.

The original presentation at the age of 21 was additionally characterised by respiratory failure and gastrointestinal involvement.

Previous treatment included cyclophosphamide, azathioprine, mycophenolate mofetil interferon alfa, and alemtuzumab. Repeat skin biopsy confirmed granulomatous infiltrates with necrotising foci and eosinophils. She failed to respond to further alemtuzumab and developed deteriorating respiratory symptoms, nasal congestion, and breast inflammation.

Rituximab was given as two infusions of mg 2 weeks apart. During the follow up period, the patient had two respiratory tract infections, which were treated with temporary increases in prednisolone dose and oral antibiotics.

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MALADIE DE CHURG ET STRAUSS PDF

EosinophiliaVasculitisAsthmaChurg-Strauss syndrome. The second stage is characterized by abnormally high numbers of eosinophils hypereosinophiliawhich causes tissue damage, most commonly to the lungs and the digestive tract. The third stage consists of vasculitis, which can eventually lead to cell death and can be life-threatening. Churg and Strauss noted three features which distinguished their patients from other patients with periarteritis nodosa but without asthma: This can cause damage to the organs from the loss of oxygen and nutrients that were being supplied by the blood.

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